https://en.wikipedia.org/wiki/Vascular_endothelial_growth_factor
Well, I guess Ed is right: "Bent legs" is actually "Bent perfusion", a combination of lung and blood perfusion.
A bent, from POV of you legs, is your own personal hypobaric chamber, UNLESS we are talking about LWB bents with upright posture and low BB. Old news, but now I can clearly see the mechanisms.
It causes mild to severe (in my case) hypoxia in your legs, which in turn trigger compensatory mechanisms via angiogenesis to lower your vascular resistance to improve perfusion. Unfortunately, this process cannot happen overnight - or any malignant tumor will kill you overnight as well.
If your vascular system is robust and flexible enough, it *adapts* and stops being a 'weak link', hence other factors (like mitochondrial density, neuronal activation strength and sheer muscle size) become dominant once again (with extra drop of recumbent-specific bracing woes on top), but if not - no matter how large your muscles are, you will not be able to pedal worth a damn if they are starved of oxygen and flooded with CO2 and lactate.
Lung function is also very important, because if your circulation is compromised, you must absolutely get the most out of it. And, obviously, if you climb higher grades, your perfusion is further compromised, because your legs get higher and heart - lower.
Unfortunately, upright posture and low BB means piss-poor aerodynamics and recumbutt unless you add fairings (that add to side area/wind load and weight).
So, what can be done?
1. Well, lots of hard training, duh. But if you want to train *smart* as well...
2. If you want to minimise your adaptation time, you should embrace the suffering, rise BB as high possible, don hypoxia mask (what's interesting about it, you should likely
wear it before and after, not during the exercise... maybe even walk around in one) .
3. Drugs that help with blood circulation and perfusion. Most of those, however, are aimed at a completely different demographic - problem with venous return and likely would make matters WORSE because 'vasoconstriction' and 'increased vascular resistance' were mentioned, but I might be completely wrong.
I suspect that such a 'performance enhancing drug' as tadalafil or sildenafil would be particularly effective for bentriders, and it is already widely used as 'gray area doping':
https://www.outsideonline.com/2105376/guide-dopings-grey-area
It is shown to be hugely effective for performance under hypoxic conditions, but not really (besides it's, ahem, prescribed effect) under normoxic.
So, I guess a trip to a local drug store is an order to test my 'bent hypoxia' theory